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Nrf2-ARE activation was significantly attenuated and the protective effects of tBHQ were reversed validating that the neuroprotective effects of Nrf2-mediated ARE activation reside in the astrocyte

You typically want to have a physical blocker like zinc oxide or titanium dioxide, she says

doi: 10.3390/ijms26094038 23 GaoL.LiuY.KimD.LiY.HwangG.NahaP

The mechanism likely involves the same angiogenic pathway: bone repair is acutely dependent on vascularization of the fracture gap, and BPC-157's VEGF-stimulating activity addresses this bottleneck directly